Stikkordarkiv: Betennelser

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Peripheral Neuropathy Fact Sheet

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Om perifer nevropati og årsaker. Nevner blodsirkulasjon og at nerven får oksygenmangel når blodkar trekker seg sammen.

http://www.ninds.nih.gov/disorders/peripheralneuropathy/detail_peripheralneuropathy.htm#183593208

«Vascular damage and blood diseases can decrease oxygen supply to the peripheral nerves and quickly lead to serious damage to or death of nerve tissues, much as a sudden lack of oxygen to the brain can cause a stroke. Diabetes frequently leads to blood vessel constriction. Various forms of vasculitis (blood vessel inflammation) frequently cause vessel walls to harden, thicken, and develop scar tissue, decreasing their diameter and impeding blood flow. This category of nerve damage, in which isolated nerves in different areas are damaged, is called mononeuropathy multiplex or multifocal mononeuropathy.»

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Complex Interaction of Sensory and Motor Signs and Symptoms in Chronic CRPS

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Nevner sammenhengen mellom nervebetennelser, stress, depresjon, m.m. i kroniske smerter. Bl.a. at «varm» vs «kald» tilstand i hånden viser til om det er i starten eller har pågått lenger enn 1 år. Den «kalde» tilstanden er mer smertefull.

http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0018775#pone-0018775-g007

«we developed a model based on interaction analysis suggesting a complex hierarchical interaction of peripheral (injury/sensory loss) and central factors (pain/disability/stress/depression) predicting motor dysfunction and hyperalgesia.»

«As the presence of an inducible autonomic nervous system autoantigen has been recently demonstrated in CRPS patients, one may speculate about possible interactions between autoimmune and inflammatory processes dominating the acute phase of CRPS, whereas in chronic CRPS the clinical signs of peripheral neurogenic inflammation and autonomic dysfunction may have mostly subsided due to post-inflammatory burn-out.»

«Furthermore, it has been demonstrated that patients initially classified as “cold” CRPS showed more pain when re-evaluated several years after the initial assessment [3]. “Warm” CRPS seems to be predominant in the acute phase of CRPS, where signs of peripheral neurogenic inflammation dominate»